There are two phases of immune response, the first response is the innate, which most times can handle things, cuts, sores etc. With bacterial and viral, ones that impact cells and turn them rogue, the innate can't defeat these antigens so easily and many times calls on the killer Ts and Bs - the adaptive immune response (the secondary, not primary response. As for the marker, if you are referring to "memory" cells, they don't really exist, that was a bit of poor science understandings from yesteryear aka assumptions.
No, cellular is not both. Are cells involved with both, always, that's how the body works, cells run the body, but there are still separate known immune responses - a primary/general first response and then a second/specialized response.
Can you elaborate on antibody dependent enhancement.
And yes, a cellular response is what happens once the innate response fails to do the job.
To say "It" what are you referring to? What is the it that makes you more susceptible? How does this work, explain the pathways.
There are two phases of immune response, the first response is the innate, which most times can handle things, cuts, sores etc. With bacterial and viral, ones that impact cells and turn them rogue, the innate can't defeat these antigens so easily and many times calls on the killer Ts and Bs - the adaptive immune response (the secondary, not primary response. As for the marker, if you are referring to "memory" cells, they don't really exist, that was a bit of poor science understandings from yesteryear aka assumptions.
No, cellular is not both. Are cells involved with both, always, that's how the body works, cells run the body, but there are still separate known immune responses - a primary/general first response and then a second/specialized response.
Asking is part of how i do research, and many times, i ask rhetorically...
You're the one that's confused - any text book will speak to innate and adaptive immune responses. Duh
You also missed the point of my whole comment on safe vs effective...